σ B contributes to Listeria monocytogenes invasion by controlling expression of inlA and inlB

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The inlA gene required for cell invasion is conserved and specific to Listeria monocytogenes.

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Listeria monocytogenes B Contributes to Invasion of Human Intestinal Epithelial Cells

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Sigma B contributes to PrfA-mediated virulence in Listeria monocytogenes.

Transcription of the Listeria monocytogenes positive regulatory factor A protein (PrfA) is initiated from either of two promoters immediately upstream of prfA (prfAp(1) and prfAp(2)) or from the upstream plcA promoter. We demonstrate that prfAp(2) is a functional sigma(B)-dependent promoter and that a sigB deletion mutation affects the virulence phenotype of L. monocytogenes. Thus, the alternat...

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The Listeria monocytogenes σB Regulon and Its Virulence-Associated Functions Are Inhibited by a Small Molecule

UNLABELLED The stress-responsive alternative sigma factor σ(B) is conserved across diverse Gram-positive bacterial genera. In Listeria monocytogenes, σ(B) regulates transcription of >150 genes, including genes contributing to virulence and to bacterial survival under host-associated stress conditions, such as those encountered in the human gastrointestinal lumen. An inhibitor of L. monocytogene...

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Listeria monocytogenes sigmaB contributes to invasion of human intestinal epithelial cells.

The role of sigma(B) in Listeria monocytogenes infection of human intestinal epithelial cells was investigated. Invasion defects associated with loss of sigma(B) paralleled those of a DeltainlA strain independently of the sigma(B)-dependent P2(prfA) promoter. Concomitantly, amounts of inlA transcript and InlA protein were significantly decreased in the DeltasigB strain.

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ژورنال

عنوان ژورنال: Microbiology

سال: 2005

ISSN: 1350-0872,1465-2080

DOI: 10.1099/mic.0.28070-0